PROSTATE CANCER PREVENTION DIET AND LIFESTYLE

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LIFESTYLE RISK FACTORS

As Director of Research at Psyche Nutrition Sciences (PNSI-Inc), it first became clear to me that nutrition and lifestyle were major risk factors for cancer more than any other preventable factors in 2003. How do we know that diet and lifestyle are the leading causes of prostate cancer? By looking to the East where the incidence is low, and dietary, lifestyle, and environmental factors are different. European and Asian immigrants adopting our way of eating and living increase their incidence of developing prostate cancer. This validates the idea that heredity plays a minor role in cancer because if genetics were the main cause, then immigrants would enjoy a lower incidence regardless of their newly adopted Western malnutrition and inactive and stress-management-barren lifestyle. More data supporting diet as a principle cause was found when researchers examined Easterners who adopted our diet in their native land. They found that people living in countries with a relatively low incidence of prostate cancer who adopt a more Western diet and lifestyle showed a higher incidence of prostate cancer.

So, what is wrong with our diet? We have a big deficiency of plant-based whole foods as well as an excess of processed foods and additives, protein, beverages, and supplements. Just as the sugar-substitute mania has not reduced the incidence of obesity, with fake and nonnutritive sugars such as Splenda, NutraSweet and Equal, the supplement explosion of recent decades has not decreased the risk of prostate cancer. In fact, there is no proof that vitamin and herbal supplements reduce prostate cancer risk. The only thing certain is that overusing supplements can result in side effects, and in some cases, disease and death. Folic acid supplements have been linked with colon cancer, and lycopene supplements have been linked with prostate cancer. Supplements may have merely provided a false sense of healthful security, enabling people to skip out on eating healthful minimally processed produce. We also may have been putting soy on an unjustifiable pedestal and been blind to other major differences between Asian and American dietary habits, such as the marked difference in amount of freshly steeped green tea consumption. Americans drink more canned and decaf tea, which are weak in, or void of, body-cleaning antioxidants. You can Americanize tea by freshly steeping green, black, white, or red tea (preferably organic) and pouring it over ice with a splash of organic apple juice—something we call TAPPLE. We also eat much more processed food, red meat, products made from flour and sugar, and much less produce.

When high-energy meals low in antioxidants and meals high in saturated fat, protein, dairy, and partially hydrogenated seed oils like canola, corn, soy, and olive, as well as fried and darkened foods are consumed over time, genetic damage from excessive free-radical formation or body pollution is thought to trigger prostate cells to begin replicating at a rapid cancerous rate.

Let’s take a look at some foods that can be placed onto a continuum regarding prostate cancer prevention. Eating certain foods regularly and over time increases the risk of developing prostate cancer. Foods and supplements that increase the risk of developing prostate cancer are called promoters. BRIGHTFOODS, at the other end of the spectrum, can decrease the risk. Medicinal foods that reduce risk are referred to as protectors. For our purposes, if a food or nutrient has insufficient data linking it to prostate cancer protection, it will be considered a promoter. Foods are not permitted to sit on the fence here and are, therefore, classified either as part of the problem or solution. Sugar substitutes, for example, are not linked to cancer production, but because they are also clearly known to have no cancer-prevention properties, they are considered prostate cancer promoters by our standards.

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